The presence of fibroids and endometrial polyps is associated with reduced endometrial receptivity and subsequent fertility outcomes. The following review explores the molecular mechanisms behind reduced endometrial receptivity, with a focus upon homebox genes, basic transcription element binding proteins, transforming growth factor β-3, and bone morphogenetic protein 2. A better understanding of these processes is essential for future targeted therapy.
J Endometr Pelvic Pain Disord 2017; 9(4): 239 - 244
Article Type: REVIEW
AuthorsShagun Narula, Amani Harris, Jim Tsaltas
- • Accepted on 24/09/2017
- • Available online on 24/10/2017
- • Published in print on 24/11/2017
This article is available as full text PDF.
- Narula, Shagun [PubMed] [Google Scholar] 1
- Harris, Amani [PubMed] [Google Scholar] 2, * Corresponding Author (email@example.com)
- Tsaltas, Jim [PubMed] [Google Scholar] 1, 2, 3
Monash University, Melbourne, Victoria - Australia
Department of Endoscopic Surgery, Monash Health, Melbourne, Victoria - Australia
Melbourne IVF, Melbourne, Victoria - Australia